Author

Richard M. Myers

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Genetics· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:statistical model· 2 Assay:ChIP-seq· 1 Research Focus:database / resource· 1 Approach:experimental assay· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

Anand VenkataramaniD· 1 shared Andrew A. HardiganiD· 1 shared Andrew Hardigan· 1 shared Anju Shukla· 1 shared Ashwin Dalal· 1 shared Atul Tandon· 1 shared Ayman W. El-Hattab· 1 shared Benjamin Cogné· 1 shared

Activity by Year

2016-2017 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperBiochemistry

Anand VenkataramanORCID

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3 papers

Recent·Most cited

GenomicsOncotargetPeer-reviewedApr 8, 2017

RNA sequencing-based cell proliferation analysis across 19 cancers identifies a subset of proliferation-informative cancers with a common survival signature

Ryne C. Ramaker, Brittany N. Lasseigne, Andrew Hardigan…·Sara J. Cooper

DOI 10.18632/oncotarget.16961

humanbulk RNA-seq

𝕏 PostBluesky

BiochemistrybioRxivPreprintMar 16, 2017

A toolbox of immunoprecipitation-grade monoclonal antibodies against human transcription factors.

Anand Venkataraman, Kun Yang, Jose Irizarry…·Seth Blackshaw

The paper develops a collection of 1406 validated monoclonal antibodies against human transcription factors and identifies factors contributing to their successful generation.

DOI 10.1101/116442

humanproteomics

𝕏 PostBluesky

GeneticsbioRxivPreprintAug 3, 2016

Mutations in EBF3 disturb transcriptional profiles and underlie a novel syndrome of intellectual disability, ataxia and facial dysmorphism

Frederike Leonie Harms, Katta Mohan Girisha, Andrew A. Hardigan…·Kerstin Kutsche

The paper identifies EBF3 mutations in individuals with intellectual disability, ataxia, and facial dysmorphism, showing they impair transcriptional regulation and neuronal development.

DOI 10.1101/067454

humanbulk RNA-seq

𝕏 PostBluesky