Author

Robert S Young

University of Edinburgh

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genomics· 1 Synthetic Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Assay:ChIP-seq· 1

Frequent Co-Authors

Christine MordsteiniD· 1 shared Grzegorz R. KudlaiD· 1 shared Jeanne BazileiD· 1 shared Juliet LuftiD· 1 shared Lana TalmaneiD· 1 shared Laurence D. HurstiD· 1 shared Martin S Taylor· 1 shared Martin S. TayloriD· 1 shared

Activity by Year

2017-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperSynthetic Biology

Christine MordsteinORCID

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2 papers

Recent·Most cited

Splicing buffers suboptimal codon usage in human cells

Christine Mordstein, Rosina Savisaar, Robert S Young, Jeanne Bazile, Lana Talmane, Juliet Luft, Michael Andre Liss, Martin S. Taylor, Laurence D. Hurst, Grzegorz R. Kudla

The paper shows splicing enhances expression of AT-rich genes by increasing mRNA stability and cytoplasmic localization, while GC-rich genes are less affected by splicing.

bioRxiv · Jan 22, 2019 · PreprintSynthetic Biology

Bidirectional transcription marks accessible chromatin and is not specific to enhancers

Robert S Young, Yatendra Kumar, Wendy A Bickmore, Martin S Taylor

The paper shows bidirectional transcription is common across all accessible chromatin regions, not just enhancers, and is not a reliable marker for enhancer activity.

Genome Biology · Dec 28, 2017Peer-reviewedGenomics