Author

S. Kawai

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

C. Alev· 1 shared H. Ohdan· 1 shared H. Tahara· 1 shared J. Toguchida· 1 shared J.-Y. Xue· 1 shared K. Fukiage· 1 shared K. Ide· 1 shared M. Nishio· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

C. Alev

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2 papers

Recent·Most cited

Development of a humanized mouse model to analyze antibodies specific for human leukocyte antigen (HLA)

S. Yanagawa, H. Tahara, T. Shirouzu, S. Kawai, Y. Tanaka, K. Ide, S. Akimoto, H. Ohdan

This paper develops a humanized mouse model to study anti-HLA antibody production, finding that allo-specific antibodies are suppressed while non-allo-specific antibodies are detectable.

PLOS ONE · Feb 5, 2021Peer-reviewedImmunology

Human iPSC-derived hypertrophic chondrocytes reveal a mutation-specific unfolded protein response in chondrodysplasias

Y. Pretemer, S. Kawai, M. Watanabe, S. Nagata, M. Nishio, S. Tamaki, C. Alev, Y. Yamanaka, J.-Y. Xue, Z. Wang, K. Fukiage, M. Tsukanaka, T. Futami, S. Ikegawa, J. Toguchida

The paper evaluates mutation-specific unfolded protein response in chondrodysplasias using human iPSC-derived hypertrophic chondrocytes and identifies differential ER stress and gene expression changes.

bioRxiv · May 19, 2020 · PreprintCell Biology