Author

J. Toguchida

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

C. Alev· 1 shared I. Taniuchi· 1 shared J.-Y. Xue· 1 shared K. Fukiage· 1 shared K. Ito· 1 shared K. Omori· 1 shared M. Nishio· 1 shared M. Tsukanaka· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

C. Alev

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

The oncogenic Runx3-Myc axis defines p53-deficient osteosarcomagenesis

S. Otani, Y. Date, T. Ueno, T. Ito, S. Kajikawa, K. Omori, I. Taniuchi, M. Umeda, T. Komori, J. Toguchida, K. Ito

This study identifies the Runx3-Myc axis as a driver of p53-deficient osteosarcomagenesis in human and mouse models.

bioRxiv · May 3, 2021 · PreprintCancer Biology

Human iPSC-derived hypertrophic chondrocytes reveal a mutation-specific unfolded protein response in chondrodysplasias

Y. Pretemer, S. Kawai, M. Watanabe, S. Nagata, M. Nishio, S. Tamaki, C. Alev, Y. Yamanaka, J.-Y. Xue, Z. Wang, K. Fukiage, M. Tsukanaka, T. Futami, S. Ikegawa, J. Toguchida

The paper evaluates mutation-specific unfolded protein response in chondrodysplasias using human iPSC-derived hypertrophic chondrocytes and identifies differential ER stress and gene expression changes.

bioRxiv · May 19, 2020 · PreprintCell Biology