Author

S. L. Stott

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1 Organism:human· 1

Frequent Co-Authors

A. I. McClatchey· 1 shared B. S. Wittner· 1 shared C. C. MacKenzie· 1 shared C. H. Liu· 1 shared D. A. Haber· 1 shared D. F. Wiley· 1 shared D. S. Micalizzi· 1 shared D. T. Miyamoto· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. I. McClatchey

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Author profile →

2 papers

Recent·Most cited

Single cell proteomics of tumor compartments identifies differential kinase activities defining sensitivity to mTOR-PI3-kinase inhibition

N. M. Karabacak, Y. Zheng, T. D. Dubash, R. Burr, D. S. Micalizzi, B. S. Wittner, D. F. Wiley, V. Comaills, E. Emmons, K. Niederhoffer, U. Ho, L. Nieman, W. Haas, S. L. Stott, D. T. Ting, , S. Maheswaran

The paper uses single cell proteomics to identify kinase activity differences in tumor compartments, showing how they affect sensitivity to mTOR-PI3K inhibition.

bioRxiv · Jan 6, 2021 · PreprintCancer Biology

Cellular mechanisms of heterogeneity in NF2-mutant schwannoma

C. C. MacKenzie, J. Vitte, C. H. Liu, E. A. Wright, E. A. Flynn, S. L. Stott, M. Giovannini, A. I. McClatchey

This study identifies intrinsic mechanisms driving heterogeneity in NF2-mutant schwannomas through analysis of mouse models and human tissue.

bioRxiv · Dec 31, 2020Cancer Biology