Author

S. n. Zhang

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:multiome· 1 Approach:statistical model· 1

Frequent Co-Authors

D. Zhang· 1 shared F.-M. Shao· 1 shared H. Cao· 1 shared J. j. Piao· 1 shared J. M. Li· 1 shared J. Y. Wang· 1 shared L. Jin· 1 shared L. Teng· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCancer Biology

D. Zhang

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2 papers

Recent·Most cited

The pan-cancer lncRNA PLANE regulates an alternative splicing program to promote cancer pathogenesis

L. Teng, Y. C. Feng, S. T. Guo, P. L. Wang, S. X. Wang, S. N. Zhang, T. F. Qi, T. La, Y. Y. Zhang, X. H. Zhao, D. Zhang, J. Y. Wang, Y. Shi, J. M. Li, H. Cao, , X. D. Zhang

This study identifies the lncRNA PLANE as a regulator of alternative splicing that promotes cancer progression by repressing NCOR2-202 splicing, leading to poor patient outcomes.

bioRxiv · Dec 4, 2020Cancer Biology

SPOCK1 promotes breast cancer progression via interacting with SIX1 and activating AKT/mTOR signaling pathway

M. Xu, X. l. Zhang, S. n. Zhang, J. j. Piao, Y. Yang, X. y. Wang, Z. h. Lin

This paper identifies SPOCK1 as a promoter of breast cancer progression by interacting with SIX1 and activating the AKT/mTOR pathway, suggesting it as a potential therapeutic target.

bioRxiv · Nov 8, 2019 · PreprintCancer Biology