Author

S. Naito

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Plant Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Assay:proteomics· 2 Organism:Arabidopsis· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1

Frequent Co-Authors

A. Balakrishnan· 1 shared A. Chatterjee· 1 shared A. Makita· 1 shared D. Pally· 1 shared D. Sidransky· 1 shared H. Onouchi· 1 shared I. Ohbayash· 1 shared K. Gondkar· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCancer Biology

A. Balakrishnan

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2 papers

Recent·Most cited

The conserved upstream ORF of the Arabidopsis ANAC082 gene mediates translational upregulation in response to nucleolar stress

S. Sasaki, T. Murakami, M. Yasumuro, A. Makita, Y. Oi, Y. Hiragori, S. Watanabe, R. Kudo, N. Hayashi, I. Ohbayash, M. Sugiyama, Y. Yamashita, S. Naito, H. Onouchi

This study shows that the upstream ORF of the Arabidopsis ANAC082 gene enhances translation under nucleolar stress but not ER stress, indicating evolutionary loss of this regulatory mechanism in ANAC103.

bioRxiv · Oct 7, 2022 · PreprintPlant Biology

Galectin-1 promotes the invasion of bladder cancer urothelia through their matrix milieu

A. Balakrishnan, D. Pally, K. Gondkar, S. Naito, D. Sidransky, A. Chatterjee, P. Kumar, R. Bhat

This study identifies Galectin-1 as a driver of bladder cancer invasion through matrix interactions, showing its role in promoting adhesion, proliferation, and mesenchymal-like phenotypes.

bioRxiv · Oct 12, 2018 · PreprintCancer Biology