Author

S. Pellegrini

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:bulk RNA-seq· 2 Approach:statistical model· 2 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

F. Michel· 2 shared Z. Li· 2 shared A. Decombe· 1 shared A. Le Tortorec· 1 shared A. Rolland· 1 shared A. Sogues· 1 shared E. Decroly· 1 shared E. Patin· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersImmunology

F. Michel

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Tick-borne flavivirus NS5 antagonizes interferon signaling by inhibiting the catalytic activity of TYK2

S. Gracias, M. Chazal, A. Decombe, Y. Unterfinger, A. Sogues, L. Pruvost, V. Robert, S. Lacour, M. Lemasson, M. Sourisseau, Z. Li, J. Richardson, E. Decroly, S. Pellegrini, V. Caval, N. Jouvenet

This study identifies a mechanism by which tick-borne flaviviruses inhibit interferon signaling by targeting TYK2 catalytic activity through their NS5 protein.

bioRxiv · Sep 7, 2023Microbiology

Human USP18 is regulated by miRNAs via the 3'UTR, a sequence duplicated in lincRNA genes residing in chr22q11.21

E. Rubino, M. Cruciani, N. Tchitchek, A. Le Tortorec, A. Rolland, O. Veli, L. Vallet, G. Gaggi, F. Michel, N. Dejucq-Rainsford, S. Pellegrini

The paper identifies miRNAs that regulate USP18 via its 3'UTR and discovers duplicated 3'UTR sequences in lincRNAs on chr22q11.21, suggesting a regulatory network for baseline USP18 levels.

bioRxiv · Oct 7, 2020Immunology

Two common disease-associated TYK2 variants impact exon splicing and TYK2 dosage

Z. Li, M. Rotival, E. Patin, F. Michel, S. Pellegrini

This study shows two TYK2 variants affect exon splicing and mildly enhance TYK2 expression, potentially contributing to autoimmune diseases.

bioRxiv · Nov 4, 2019Immunology