Author

S. Saijou

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cancer Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Y. Matsumura· 3 shared M. Yasunaga· 2 shared S. Hanaoka· 2 shared T. Anzai· 2 shared H. Kurosawa· 1 shared H. Takashima· 1 shared M. Hara· 1 shared Y. Ohnuki· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

8 top collaborators · 22 mutual co-author links

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Curved chords = mutual co-authorship

3 shared papersBiophysics

Y. Matsumura

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Identification of CD73 as the antigen of an antigen-unknown monoclonal antibody established by exosome immunization, and its antibody-drug conjugate exerts an antitumor effect on glioblastoma cell lines

T. Anzai, S. Saijou, H. Takashima, M. Hara, S. Hanaoka, Y. Matsumura, M. Yasunaga

This paper identifies CD73 as the antigen of a monoclonal antibody generated by exosome immunization and shows its antibody-drug conjugate has antitumor effects on glioblastoma cell lines.

bioRxiv · Sep 30, 2021 · PreprintBiophysics

A novel and potent thrombolytic fusion protein consisting of anti-insoluble fibrin antibody and mutated urokinase

S. Hanaoka, S. Saijou, Y. Matsumura

This paper develops a thrombolytic fusion protein, AMU1114, that selectively targets insoluble fibrin, reducing systemic side effects compared to tPA and UK.

bioRxiv · Sep 6, 2020Pharmacology & Toxicology

TMEM180 contributes to colorectal cancer proliferation through intracellular metabolic pathways

T. Anzai, S. Saijou, Y. Ohnuki, H. Kurosawa, M. Yasunaga, Y. Matsumura

This study shows TMEM180 promotes colorectal cancer growth by enhancing glucose and glutamine metabolism, despite not affecting oxygen consumption or phosphorylation.

bioRxiv · Jul 16, 2020Cancer Biology