Author

S. Vanderschueren

Universitair Ziekenhuis Leuven

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell-cell communication· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:flow cytometry· 1

Frequent Co-Authors

A. Damerau· 1 shared A. Hombrouck· 1 shared Adrian ListoniD· 1 shared B. Pillay· 1 shared Bruno FautreliD· 1 shared Carine Helena WoutersiD· 1 shared Christophe PouletiD· 1 shared D. Cassiman· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperImmunology

A. Damerau

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Adult patients with autoinflammation of unknown origin partially phenocopy the immune presentation of Still's disease

Rafael Veiga, Leana De Vuyst, Christophe Poulet, Julika Neumann, Leoni Bücken, Teresa Prezzemolo, Mathijs Willemsen, S. Vanderschueren, Patrick Matthys, Emna Chabaane, Maximilien Fléron, Gaël Cobraiville, Dominique Baiwir, Gabriel Mazzucchelli, Bruno Fautrel, , Adrian Liston

The paper compares immunological profiles of patients with autoinflammation of unknown origin to healthy controls and Still's disease patients, identifying shared biomarkers and immune features.

Nature Communications · Apr 1, 2026Peer-reviewedImmunology

Human ADA2 deficiency is characterized by the absence of an intracellular hypoglycosylated form of adenosine deaminase 2

L. Ehlers, M. Wouters, A. Hombrouck, B. Pillay, S. Delafontaine, G. Bucciol, M. Baggio, M. Dzhus, E. Frances Rabanal, A. Damerau, J. Neugebauer, F. Ebstein, M. Jacquemyn, L. De Somer, R. Schrijvers, , I. Meyts

The paper identifies an intracellular hypoglycosylated form of ADA2 absent in DADA2 patients, linking its absence to reduced deaminase activity and variant pathogenicity.

bioRxiv · Oct 25, 2023 · PreprintImmunology