Author

Santosh K. Maurya

The Ohio State University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Physiology· 1

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Ahlke HeydemanniD· 1 shared Amy WebbiD· 1 shared Andrew N. CarleyiD· 1 shared Azariyas A. ChallaiD· 1 shared Chandan Kumar MauryaiD· 1 shared Christos P. KyriakopoulosiD· 1 shared Craig H. SelzmaniD· 1 shared E. Douglas LewandowskiiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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1 shared paperPhysiology

Ahlke HeydemannORCID

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1 paper

Recent·Most cited

Increased CPT1a expression is a critical cardioprotective response to pathological stress that suppresses gene programs for remodeling and enables rescue by gene transfer

Andrew N. Carley, Santosh K. Maurya, Chandan Kumar Maurya, Yang Wang, Amy Webb, Azariyas A. Challa, Tatiana Gromova, Thomas M. Vondriska, Zhentao Zhang, Hua Zhu, Ahlke Heydemann, Kenneth C. Bedi, Christos P. Kyriakopoulos, Craig H. Selzman, Stavros George Drakos, , E. Douglas Lewandowski

This study shows that increased CPT1a expression protects the heart from pathological stress by suppressing harmful gene programs, and gene transfer can rescue cardiac function in disease models.

Circulation Research · Dec 4, 2025Peer-reviewedPhysiology