Author

Hua Zhu

The Ohio State UniversityORCID 0000-0001-7136-7326Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Hua Zhu2 papers · University of Pennsylvania

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Ahlke HeydemanniD· 1 shared Aidan C. ManningiD· 1 shared Alejandra Patino-GuerreroiD· 1 shared Amy WebbiD· 1 shared Andrew N. CarleyiD· 1 shared Azariyas A. ChallaiD· 1 shared Chandan Kumar MauryaiD· 1 shared Christos P. KyriakopoulosiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperPhysiology

Ahlke HeydemannORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

A tRNA-derived small RNA orchestrates cardioprotection through distinct cell-type-specific mechanisms

Tian Hao, Jin Li, Kaushik V Amancherla, Lingfei Sun, Priyanka Gokulnath, Alejandra Patino-Guerrero, Ritin Sharma, Krystine Garcia‐Mansfield, Chunyang Xiao, Aidan C. Manning, Emeli Chartterjee, Xiaoting Yu, Richard Xu, Yiming Zhang, Zhihong Yang, , Guoping Li

This study identifies a tRNA-derived small RNA that reduces cardiac fibrosis and hypertrophy through cell-type-specific mechanisms in mouse and human cardiac cells.

bioRxiv · Sep 29, 2026 · PreprintCell Biology

Increased CPT1a expression is a critical cardioprotective response to pathological stress that suppresses gene programs for remodeling and enables rescue by gene transfer

Andrew N. Carley, Santosh K. Maurya, Chandan Kumar Maurya, Yang Wang, Amy Webb, Azariyas A. Challa, Tatiana Gromova, Thomas M. Vondriska, Zhentao Zhang, Hua Zhu, Ahlke Heydemann, Kenneth C. Bedi, Christos P. Kyriakopoulos, Craig H. Selzman, Stavros George Drakos, , E. Douglas Lewandowski

This study shows that increased CPT1a expression protects the heart from pathological stress by suppressing harmful gene programs, and gene transfer can rescue cardiac function in disease models.

Circulation Research · Dec 4, 2025Peer-reviewedPhysiology