Author

Shachi Salvi

University of Michigan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Developmental Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

Anshika SrivastavaiD· 2 shared BT McGrathiD· 2 shared Stephanie L. BielasiD· 2 shared Yao-Chang TsaniD· 2 shared Adam S. HelmsiD· 1 shared Amanda MocciaiD· 1 shared CE Keegan· 1 shared Donna M. MartiniD· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersDevelopmental Biology

Anshika SrivastavaORCID

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2 papers

Recent·Most cited

Aberrant extracellular matrix and cardiac development in models lacking the PR-DUB component ASXL3

BT McGrath, Yao-Chang Tsan, Shachi Salvi, Neeti Ghali, Donna M. Martin, Mark Hannibal, CE Keegan, Adam S. Helms, Anshika Srivastava, Stephanie L. Bielas

This study investigates the role of ASXL3 in cardiac development using mouse and human models, finding that its loss leads to cardiac abnormalities and extracellular matrix dysfunction.

bioRxiv · Jul 14, 2022 · PreprintDevelopmental Biology

ASXL3 controls cortical neuron fate specification through extrinsic self-renewal pathways

BT McGrath, P Wu, Shachi Salvi, Navjit S Girgla, Xin Chen, Jiaqiang Zhu, R KC, Yao-Chang Tsan, Amanda Moccia, Anshika Srivastava, X Zhou, Stephanie L. Bielas

This study shows ASXL3 regulates cortical neuron fate through extrinsic signaling and epigenetic pathways, linking it to ASD risk.

bioRxiv · Jul 20, 2021 · PreprintNeuroscience