Author

T. A. Partridge

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

J. S. Novak· 2 shared B. Chazaud· 1 shared B. T. Harris· 1 shared C. T. Rossi· 1 shared D. A. Mazala· 1 shared D. Alladi· 1 shared D. Ashley Hill· 1 shared D. Mazala· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersPathology

J. S. Novak

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2 papers

Recent·Most cited

Altered muscle niche contributes to myogenic deficit in the D2-mdx model of severe DMD

D. A. Mazala, R. Hindupur, Y. J. Moon, F. Shaikh, I. H. Gamu, D. Alladi, G. Panci, M. Weiss-Gayet, B. Chazaud, T. A. Partridge, J. S. Novak, J. K. Jaiswal

This study shows that altered muscle niche in D2-mdx mice leads to poor myogenesis and increased degeneration, but reversing this improves regeneration in adult mice.

bioRxiv · Mar 27, 2023 · PreprintPathology

Human muscle stem cells are refractory to aging

J. S. Novak, D. Mazala, M. Nearing, N. F. Habib, T. Dickson, O. B. Ioffe, B. T. Harris, M. N. Fidelia-Lambert, C. T. Rossi, D. Ashley Hill, K. R. Wagner, E. P. Hoffman, T. A. Partridge

This study shows that human muscle stem cells retain myogenic capacity across age and postmortem time, suggesting age-related muscle loss is due to environmental factors rather than intrinsic cell dysfunction.

Aging Cell · Jun 5, 2021Peer-reviewedCell Biology