Author

T. A. Quinn

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 4

Signature Assays, Organisms & Methods

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Assay:electrophysiology· 4 Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 4 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1 Assay:imaging· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

B. A. Cameron· 3 shared P. Kohl· 3 shared J. Greiner· 2 shared M. R. Stoyek· 2 shared R. Peyronnet· 2 shared B. Han· 1 shared C. Zgierski-Johnston· 1 shared E. A. DeLong· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersPhysiology

B. A. Cameron

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Repolarisation-relaxation dyscoupling and TRPA1 activation permit systolic mechano-arrhythmogenesis

B. A. Cameron, M. R. Stoyek, J. Greiner, R. Peyronnet, P. Kohl, T. A. Quinn

This study shows that TRPA1 activation and disrupted repolarisation-relaxation coupling during systole contribute to mechano-arrhythmogenesis in ischaemic conditions.

bioRxiv · Feb 21, 2024 · PreprintPhysiology

Perivascular Excitation Tunnelling: a Novel and Preventable Cause of Cardiac Reperfusion Arrhythmias

E. Chleilat, T. Puig Walz, B. Han, T. A. Quinn, P. Kohl, C. Zgierski-Johnston

This study identifies perivascular excitation tunnelling (PVET) as a novel mechanism causing reperfusion arrhythmias and shows that two-step reperfusion can prevent it in rabbit and pig hearts.

bioRxiv · Nov 29, 2023 · PreprintPhysiology

Mechano-arrhythmogenicity is enhanced during late repolarisation in ischemia and driven by a TRPA1-, calcium-, and reactive oxygen species-dependent mechanism

B. A. Cameron, T. A. Quinn

This study identifies that mechano-arrhythmogenicity in ischemia is heightened during late repolarisation and mediated by TRPA1, calcium, and reactive oxygen species.

bioRxiv · Aug 20, 2021 · PreprintPhysiology

The cardiac TRPA1 channel drives calcium-mediated mechano-arrhythmogenesis

B. A. Cameron, M. R. Stoyek, J. J. Bak, M. S. Connolly, E. A. DeLong, J. Greiner, R. Peyronnet, P. Kohl, T. A. Quinn

The paper shows TRPA1 channels drive calcium-mediated mechano-arrhythmogenesis in cardiac cells under conditions of altered repolarization-relaxation coupling.

bioRxiv · Oct 1, 2020 · PreprintPhysiology