Author

T. Alliston

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Bioengineering· 1 Cancer Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Organism:human· 2 Assay:imaging· 2 Assay:proteomics· 2 Approach:deep learning· 1

Frequent Co-Authors

C. A. Schurman· 2 shared A. Dang· 1 shared A. J. Fields· 1 shared A. Kuo· 1 shared A. O'Broin· 1 shared A. R. Marks· 1 shared A. Robling· 1 shared B. A. Hain· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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2 shared papersBioengineering

C. A. Schurman

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Author profile →

4 papers

Recent·Most cited

Deep learning models to map osteocyte networks can successfully distinguish between young and aged bone

S. Dreyer Vetter, C. A. Schurman, T. Alliston, G. Slabaugh, S. W. Verbruggen

This study uses deep learning to segment osteocyte networks and shows it can distinguish between young and aged mouse bones.

bioRxiv · Dec 20, 2023Bioengineering

Zoledronic acid improves bone quality and muscle function in a high bone turnover state

T. Trivedi, M. Manaa, S. John, S. Reiken, S. Murthy, G. M. Pagnotti, N. S. Dole, Y. She, S. Suresh, B. A. Hain, J. Regan, R. Ofer, L. Wright, A. Robling, X. Cao, , T. A. Guise

This study shows zoledronic acid improves bone quality and muscle function in a mouse model of metabolic bone disease by reducing TGFβ release from bone.

bioRxiv · Jun 1, 2023 · PreprintCancer Biology

Deep Coverage and Quantification of the Bone Proteome Provides Enhanced Opportunities for New Discoveries in Skeletal Biology and Disease

J. P. Rose, C. A. Schurman, C. D. King, J. Bons, J. B. Burton, S. K. Patel, A. O'Broin, T. Alliston, B. Schilling

This paper introduces a novel mass spectrometric workflow for deep bone proteome coverage and quantification, identifying key proteins and post-translational modifications relevant to bone aging and disease.

bioRxiv · Nov 20, 2022 · PreprintBiochemistry

Suppressed Osteocyte Perilacunar / Canalicular RemodelingPlays a Causal Role in Osteoarthritis

C. M. Mazur, J. J. Woo, C. S. Yee, A. J. Fields, C. Acevedo, K. N. Bailey, T. W. Fowler, J. C. Lotz, A. Dang, A. Kuo, T. P. Vail, T. Alliston

This study shows that suppressed osteocyte perilacunar/canalicular remodeling causes osteoarthritis by impairing cartilage homeostasis in humans and mice.

bioRxiv · Jan 31, 2019Physiology