Author

T. Simon-Vermot

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:zebrafish· 3 Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

R. M. White· 3 shared A. Baggiolini· 2 shared B. S. Taylor· 2 shared J. M. Weiss· 2 shared L. Studer· 2 shared N. R. Campbell· 2 shared R. Koche· 2 shared A. A. Jungbluth· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersCancer Biology

R. M. White

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3 papers

Recent·Most cited

Bioluminescent zebrafish transplantation model for drug discovery

M. Hason, J. Jovicic, I. Vonkova, M. Bojic, T. Simon-Vermot, R. M. White, P. Bartunek

This study develops a bioluminescent zebrafish model to screen kinase inhibitors for cancer therapy, finding that NanoLuc luciferase enables sensitive and rapid in vivo drug response evaluation.

bioRxiv · Mar 3, 2022Pharmacology & Toxicology

Anatomic position determines oncogenic specificity in melanoma

J. M. Weiss, M. V. Hunter, N. M. Cruz, A. Baggiolini, M. Tagore, Y. Ma, S. Misale, M. Marasco, T. Simon-Vermot, N. R. Campbell, F. Newell, J. S. Wilmott, P. A. Johansson, J. F. Thompson, G. V. Long, , R. M. White

The paper shows that anatomic position influences oncogenic specificity in melanoma by identifying position-specific gene programs that interact with oncogenes like CRKL.

bioRxiv · Nov 14, 2020Cancer Biology

Developmental chromatin programs determine oncogenic competence in melanoma

A. Baggiolini, S. J. Callahan, T. Trieu, M. M. Tagore, E. Montal, J. M. Weiss, S. E. Tischfield, Y. Fan, N. R. Campbell, N. Saurat, T. Hollmann, T. Simon-Vermot, S. K. Tickoo, B. S. Taylor, R. Koche, , R. M. White

The paper shows that BRAFV600E transforming ability depends on the cell of origin's transcriptional program, with neural crest and melanoblasts being more susceptible due to higher chromatin modifying enzyme expression and ATAD2-SOX10 complex formation.

bioRxiv · May 9, 2020Cancer Biology