Author

J. M. Weiss

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 4

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Assay:bulk RNA-seq· 3 Research Focus:cancer & tumor microenvironment· 3 Organism:zebrafish· 3 Research Focus:cell type annotation· 2 Organism:human· 2 Research Focus:cell-cell communication· 1 Assay:ChIP-seq· 1

Frequent Co-Authors

R. M. White· 3 shared A. Baggiolini· 2 shared B. S. Taylor· 2 shared L. Studer· 2 shared M. V. Hunter· 2 shared N. R. Campbell· 2 shared R. Koche· 2 shared T. Simon-Vermot· 2 shared

Activity by Year

2019-2020 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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3 shared papersCancer Biology

R. M. White

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Author profile →

4 papers

Recent·Most cited

Anatomic position determines oncogenic specificity in melanoma

J. M. Weiss, M. V. Hunter, N. M. Cruz, A. Baggiolini, M. Tagore, Y. Ma, S. Misale, M. Marasco, T. Simon-Vermot, N. R. Campbell, F. Newell, J. S. Wilmott, P. A. Johansson, J. F. Thompson, G. V. Long, , R. M. White

The paper shows that anatomic position influences oncogenic specificity in melanoma by identifying position-specific gene programs that interact with oncogenes like CRKL.

bioRxiv · Nov 14, 2020Cancer Biology

Spatially resolved transcriptomics reveals the architecture of the tumor/microenvironment interface

M. V. Hunter, R. Moncada, J. M. Weiss, I. Yanai, R. M. White

This study uses spatial transcriptomics to identify a conserved tumor-microenvironment interface in zebrafish and human melanoma characterized by cilia gene upregulation.

bioRxiv · Nov 5, 2020Cancer Biology

Developmental chromatin programs determine oncogenic competence in melanoma

A. Baggiolini, S. J. Callahan, T. Trieu, M. M. Tagore, E. Montal, J. M. Weiss, S. E. Tischfield, Y. Fan, N. R. Campbell, N. Saurat, T. Hollmann, T. Simon-Vermot, S. K. Tickoo, B. S. Taylor, R. Koche, , R. M. White

The paper shows that BRAFV600E transforming ability depends on the cell of origin's transcriptional program, with neural crest and melanoblasts being more susceptible due to higher chromatin modifying enzyme expression and ATAD2-SOX10 complex formation.

bioRxiv · May 9, 2020Cancer Biology

TGFβ blocks IFNα/β release and tumor rejection in spontaneous mammary tumors

M. V. V. Guerin, F. Regnier, V. Feuillet, L. Vimeux, J. M. Weiss, G. Bismuth, G. Altan-Bonnet, T. Guilbert, M. Thoreau, V. Finisguerra, E. Donnadieu, A. Trautmann, N. Bercovici

This study shows that TGFβ inhibits IFNα/β production and tumor rejection in spontaneous mammary tumors, and blocking TGFβ enhances DMXAA-induced tumor regression.

bioRxiv · Jul 9, 2019Cancer Biology