Author

Takashi Okumura

The University of Texas MD Anderson Cancer Center

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Organism:Drosophila· 1

Frequent Co-Authors

Aatur D. Singhi· 1 shared Akiko SagaraiD· 1 shared Anirban MaitraiD· 1 shared Anirban Maitra· 1 shared Benson Chellakkan SelvanesaniD· 1 shared Bidyut Ghosh· 1 shared Catherine G Fisher· 1 shared Fredrik I. ThegeiD· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

Aatur D. Singhi

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Cancer BiologyGutPeer-reviewedSep 13, 2024

Metabolic Reprogramming by Mutant GNAS Creates an Actionable Dependency in Intraductal Papillary Mucinous Neoplasms of the Pancreas

Yuki Makino, Kimal Rajapakshe, Benson Chellakkan Selvanesan…·Fredrik I. Thege

This study shows that mutant GNAS drives a gastric metaplasia gene signature and glycolytic dependency in IPMNs, offering a therapeutic target.

16 citations

mousebulk RNA-seq

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Developmental BiologyDevelopmentPeer-reviewedMar 2, 2023

The Drosophila AWP1 ortholog Doctor No regulates JAK/STAT signaling for left-right asymmetry in the gut by promoting receptor endocytosis

Yi‐Ting Lai, Takeshi Sasamura, Junpei Kuroda…·Kenji Matsuno

5 citations

Drosophilastatistical model

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Cancer BiologybioRxivPreprintApr 28, 2018

GnasR201C Induces Murine Pancreatic Cystic Neoplasms through Suppression of YAP1 Signaling and Transcriptional Reprogramming

Noboru Ideno, Hiroshi Yamaguchi, Bidyut Ghosh…·Anirban Maitra

This study shows that GnasR201C induces pancreatic cystic neoplasms in mice by suppressing YAP1 signaling and reprogramming transcription towards a differentiated phenotype.

DOI 10.1101/310292

mouseexperimental assay

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