Author

Benson Chellakkan Selvanesan

The University of Texas MD Anderson Cancer CenterORCID 0000-0001-8013-1294Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cancer Biology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Assay:bulk RNA-seq· 2 Organism:human· 2 Assay:spatial transcriptomics· 2 Approach:deep learning· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1

Frequent Co-Authors

Anirban MaitraiD· 3 shared Jimin MiniD· 2 shared Akiko SagaraiD· 1 shared Ana Vilchis Celis· 1 shared Andreas MundiD· 1 shared Benjamin J. SwansoniD· 1 shared Bongjun KimiD· 1 shared Brian W. Wolpin· 1 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersCancer Biology

Anirban MaitraORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cancer BiologybioRxivPreprintOct 6, 2026

KRAS-driven metabolic reprogramming of enhanced MFSD2A-mediated lysophosphatidylcholine scavenging promotes survival and progression of pancreatic ductal adenocarcinoma

Yihui Chen, Cason Haffner, Benson Chellakkan Selvanesan…·Johannes Francois Fahrmann

This study identifies KRAS-driven metabolic reprogramming through MFSD2A-mediated lysophosphatidylcholine scavenging as a driver of PDAC progression and shows that targeting this pathway with edelfosine has anti-cancer effects.

DOI 10.64898/2026.10.05.756824

humanbulk RNA-seq

𝕏 PostBluesky

Cancer BiologyCancer DiscoveryPeer-reviewedApr 21, 2026

AI-powered Deep Visual Proteomics reveals critical molecular transitions in pancreatic cancer precursors

Jimin Min, Lisa Schweizer, Gijs Zonderland…·Anirban Maitra

This study uses AI-driven proteomics to identify molecular transitions in pancreatic cancer precursors, revealing early molecular changes in histologically normal tissue.

7 citationstop 1%

humanproteomics

𝕏 PostBluesky

Cancer BiologyGutPeer-reviewedSep 13, 2024

Metabolic Reprogramming by Mutant GNAS Creates an Actionable Dependency in Intraductal Papillary Mucinous Neoplasms of the Pancreas

Yuki Makino, Kimal Rajapakshe, Benson Chellakkan Selvanesan…·Fredrik I. Thege

This study shows that mutant GNAS drives a gastric metaplasia gene signature and glycolytic dependency in IPMNs, offering a therapeutic target.

16 citations

mousebulk RNA-seq

𝕏 PostBluesky