Author

Taylor Kaye

Ashland University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Developmental Biology· 2

Signature Assays, Organisms & Methods

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Organism:zebrafish· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

Mason PosneriD· 2 shared Stuart Brdicka· 2 shared Adil S. Hussen· 1 shared Alexis Roberts· 1 shared Brandon Andrew· 1 shared Bryce Patterson· 1 shared Dylan R. FarnsworthiD· 1 shared Emmaline Kepp· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersDevelopmental Biology

Mason PosnerORCID

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2 papers

Recent·Most cited

Loss of αBa-crystallin, but not αA-crystallin, increases age-related cataract in the zebrafish lens

Mason Posner, Taylor Garver, Taylor Kaye, Stuart Brdicka, Madison Suttle, Bryce Patterson, Dylan R. Farnsworth

This study shows that loss of zebrafish Ba-crystallin (cryaba) increases age-related cataract, while loss of A-crystallin (cryaa) does not, highlighting an ontogenetic shift in crystallin function during lens development.

Experimental Eye Research · Jul 1, 2024Peer-reviewedDevelopmental Biology

Effects of α-crystallin gene knockout on zebrafish lens development

Mason Posner, Kelly L. Murray, Brandon Andrew, Stuart Brdicka, Alexis Roberts, Kirstan Franklin, Adil S. Hussen, Taylor Kaye, Emmaline Kepp, Mathew S. McDonald, Tyler Snodgrass, Keith D. Zientek, Larry L. David

This study investigates the effects of α-crystallin gene knockout on zebrafish lens development, finding that loss of A-crystallin causes lens defects while loss of Ba- or Bb-crystallin does not.

bioRxiv · Dec 22, 2021 · PreprintDevelopmental Biology