the Kidney Precision Medicine Project

The paper compares miRNA-mRNA profiles across human and murine AKI models to identify shared and specific molecular programs associated with different injury etiologies and biological heterogeneity.

The paper uses ontologies to integrate clinical, pathological, and molecular kidney data for precision medicine analysis.

The paper integrates single-cell and microdissected transcriptomes to show fructose transporter expression varies across rat nephron segments, with S3 being the primary site of fructose reabsorption.

This study identifies conserved myeloid subsets and disease-associated gene expression changes in kidney injury across lupus mouse models and human patients with lupus nephritis.

This study maps the spatial transcriptomic landscape of the human kidney papilla in stone disease, identifying immune injury and matrix remodeling as key features.