Author

W. G. Ambler

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

N. Schwartz· 2 shared T. T. Lu· 2 shared W. D. Shipman· 2 shared Y. Chinenov· 2 shared A. Daamen· 1 shared A. Jabbari· 1 shared A. Rathod· 1 shared B. Mehrara· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersImmunology

N. Schwartz

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2 papers

Recent·Most cited

ImmunologybioRxivJun 13, 2022

Lymphatic dysfunction in lupus contributes to cutaneous photosensitivity and lymph node B cell responses

M. J. Howlader, W. G. Ambler, M. L. S. Chalasani…·T. T. Lu

This study shows that lymphatic dysfunction in lupus exacerbates cutaneous photosensitivity and lymph node B cell responses, and that improving lymphatic flow reduces these effects.

DOI 10.1172/jci168412

humanmouse

𝕏 PostBluesky

ImmunologybioRxivAug 18, 2021

The interferon-rich skin environment regulates Langerhans cell ADAM17 to promote photosensitivity in lupus

T. M. Li, V. Zyulina, E. S. Seltzer…·T. T. Lu

This study shows that IFN-I in lupus skin reduces Langerhans cell ADAM17 activity, leading to increased photosensitivity, and that anti-IFNAR treatment restores ADAM17 function and reduces photosensitivity.

DOI 10.7554/elife.85914

humanmouse

𝕏 PostBluesky