N-terminal acetylation reduces α-synuclein pathology in models of Parkinson's disease
Esther del Cid‐Pellitero, Thomas Goiran, Zaid A.M. Al‐Azzawi, Nathan C. Karpilovsky, Jace Jones-Tabah, Jonas J. Mayo, Frédérique Larroquette, Wen Luo, Irina Shlaifer, Yogitha Thattikota, Andrew N. Bayne, Jean‐François Trempe, Thomas M. Durcan, Edward A. Fon
This study shows that N-terminal acetylation of α-synuclein reduces aggregation, uptake, seeding, and toxicity in Parkinson's disease models.
Neurobiology of Disease · Jun 11, 2026Peer-reviewedMolecular Biology