Author

Renato C. Monteiro

Centre National de la Recherche ScientifiqueORCID 0000-0001-5202-5646Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 2 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:GWAS / association mapping· 1 Assay:GWAS summary statistics· 1

Frequent Co-Authors

Julie Bex-CoudratiD· 2 shared Adele MitrottiiD· 1 shared Aftab S. ChishtiiD· 1 shared Agnès CosteiD· 1 shared Akchurin Oleh· 1 shared Alejandro Quiroga· 1 shared Alexandra CambieriD· 1 shared Amandine Badie· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersImmunology

Julie Bex-CoudratORCID

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3 papers

Recent·Most cited

ImmunologyThe Journal of Experimental MedicinePeer-reviewedMar 26, 2026

Basophils drive the resolution and promote wound healing in adult and aged mice

Julie Bex-Coudrat, Victoria Peter, Chaimae Saji…·Christophe Pellefigues

This study shows basophils promote inflammation resolution and wound healing in both adult and aged mice by driving tissue remodeling and immunoregulatory functions.

5 citationstop 1%

mousebulk RNA-seq

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ImmunologyPLoS ONEPeer-reviewedOct 7, 2025

Blockade of mTOR ameliorates IgA Nephropathy by correcting CD89 and CD71 dysfunctions in humanized mice

Alexandra Cambier, Jennifer Da Silva, Julie Bex-Coudrat…·Renato C. Monteiro

This study shows that blocking mTOR reduces IgA nephropathy in humanized mice by correcting CD89 and CD71 dysfunctions and decreasing immune complex deposits.

4 citations

mousebulk RNA-seq

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Genetic & Genomic MedicinemedRxivPreprintNov 19, 2021

GWAS defines pathogenic signaling pathways and prioritizes drug targets for IgA nephropathy

Krzysztof Kiryluk, Elena Sánchez, Shu‐Feng Zhou…·Claudia Izzi

This study identifies 30 genome-wide significant risk loci for IgA nephropathy, links them to inflammatory pathways, and prioritizes drug targets.

5 citations

humanGWAS summary statistics

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