Author

Soumya Mukherjee

Washington University in St. LouisORCID 0000-0001-5326-4008Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:proteomics· 3 Research Focus:differential expression· 2 Approach:statistical model· 2 Research Focus:benchmarking· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1

Frequent Co-Authors

Chihiro SatoiD· 2 shared Kaleigh F. RobertsiD· 2 shared Randall John BatemaniD· 2 shared Reid A. CoyleiD· 2 shared Srinivas KoutarapuiD· 2 shared Alison GoateiD· 1 shared Blaine Russell RobertsiD· 1 shared Catriona McLeaniD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersNeuroscience

Chihiro SatoORCID

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3 papers

Recent·Most cited

NeuroscienceAnnals of NeurologyPeer-reviewedJul 19, 2026

Distribution of big tau isoforms in the human central and peripheral nervous system

Rama K. Koppisetti, Nicolas R. Barthélemy, Kanta Horie…·Soumya Mukherjee

The paper characterizes big tau isoforms in human nervous system tissues and CSF, finding they are enriched in the peripheral nervous system and largely unaffected by neurodegenerative disease.

7 citationstop 1%

humanproteomics

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NeuroscienceActa NeuropathologicaPeer-reviewedJul 22, 2025

Isomerized Aβ in the brain can distinguish the status of amyloidosis in the Alzheimer's Disease spectrum

Soumya Mukherjee, Reid A. Coyle, Céline Dubois…·Blaine Russell Roberts

The paper quantifies post-translational modifications of Aβ in brain tissue to distinguish amyloidosis stages in Alzheimer's Disease and related conditions.

5 citations

humanproteomics

𝕏 PostBluesky

NeurosciencebioRxivPreprintJun 11, 2025

Post-translational modifications distinguish amyloid-β isoform patterns extracted from vascular deposits and parenchymal plaques

Srinivas Koutarapu, Kaleigh F. Roberts, Reid A. Coyle…·Soumya Mukherjee

The paper identifies differences in post-translational modifications of amyloid-β isoforms between vascular deposits and parenchymal plaques in Alzheimer's disease.

DOI 10.1101/2025.06.11.658821

humanproteomics

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