Author

Kameswaran Surendran

University of South DakotaORCID 0000-0001-5693-4409Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Organism:mouse· 2 Approach:statistical model· 2 Approach:experimental assay· 1

Frequent Co-Authors

Jennifer deRiso· 2 shared Madhusudhana R. JangaiD· 2 shared Malini Mukherjee· 2 shared Alicia Simmons· 1 shared Eric Fogarty· 1 shared Indra ChandrasekariD· 1 shared Jianning TaoiD· 1 shared Michael S. KaretaiD· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 24 mutual co-author links

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2 shared papersDevelopmental Biology

Jennifer deRiso

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2 papers

Recent·Most cited

Kidney collecting duct cell type composition is regulated by Notch signaling via modulation of mTORC1

Jennifer deRiso, Malini Mukherjee, Madhusudhana R. Janga, Alicia Simmons, Michael S. Kareta, Jianning Tao, Indra Chandrasekar, Kameswaran Surendran

This study identifies Hes1 as a Notch signaling target that regulates mTORC1 activity to maintain distinct epithelial cell types in kidney collecting ducts.

bioRxiv · Apr 9, 2024 · PreprintDevelopmental Biology

Foxi1 inactivation rescues loss of principal cell fate selection in Hes1-deficient kidneys but does not ensure maintenance of principal cell gene expression.

Malini Mukherjee, Jennifer deRiso, Madhusudhana R. Janga, Eric Fogarty, Kameswaran Surendran

This study shows that Hes1 represses Foxi1 to ensure principal cell fate selection in kidney collecting ducts, but Foxi1 inactivation does not fully restore principal cell gene expression.

Developmental Biology · Aug 13, 2020Peer-reviewedDevelopmental Biology