Author

Katherine L. Cotten

Johns Hopkins UniversityORCID 0000-0001-5814-2093Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:bacteria (other)· 1 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Kimberly Michele DavisiD· 2 shared Abigail E. McShane· 1 shared Ashley Augustiny ChapiniD· 1 shared Justin Greene· 1 shared Justin M. StineiD· 1 shared Peter C. DedoniD· 1 shared Reza Ghodssi AiD· 1 shared Rezia Era D. BrazaiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersMicrobiology

Kimberly Michele DavisORCID

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2 papers

Recent·Most cited

MicrobiologyPLoS PathogensPeer-reviewedJul 7, 2025

Yersinia pseudotuberculosis growth arrest during type-III secretion system expression is associated with altered ribosomal protein expression and decreased gentamicin susceptibility

Justin Greene, Rhett A. Snyder, Katherine L. Cotten…·Kimberly Michele Davis

This study shows that Yersinia pseudotuberculosis growth arrest during T3SS expression reduces ribosomal protein levels and increases gentamicin tolerance.

4 citations

mousebulk RNA-seq

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MicrobiologybioRxivPreprintJul 1, 2025

Loss of the s2U tRNA modification induces antibiotic tolerance and is linked to changes in ribosomal protein expression

Katherine L. Cotten, Abigail E. McShane, Peter C. Dedon…·Kimberly Michele Davis

The paper shows that loss of s2U tRNA modification in Yersinia pseudotuberculosis leads to antibiotic tolerance by reducing ribosomal protein translation.

DOI 10.1101/2025.07.01.662621

bacteria (other)proteomics

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