Jia-Ying Su

This study investigates how Hcp in the T6SS selectively delivers diverse effectors using structural and functional analyses.

The paper identifies UA dinucleotides as a major determinant of RNA stability through UTR variants, linking them to disease and immune response pathways.

The paper analyzes 292 disease-relevant deleterious variants in 486,956 participants to assess their risk differences, finding most have minimal impact.

This study identifies UTR mutations that disrupt translation by altering polysome profiles and RNA structures, with a focus on uORF initiation sites and SRSF2 binding motifs.