Blandine M. D. Franke-Fayard

This study investigates the evolutionary dynamics and essential function of pir genes in malaria parasites, identifying pirC1 as critical for blood stage growth and suggesting a role in host cell interaction or nutrient uptake.

This study examines humoral and cellular immunity to genetically attenuated malaria parasites in humans and identifies key liver-stage antigens for vaccine development.

The paper identifies EMAP3 as a new parasite protein exposed on the surface of P. berghei infected red blood cells, which interacts with EMAP1 but is not the main CD36-binding protein.