Dillon E Sloan

The paper investigates how CCDC32 interacts with AP-2 to regulate its assembly, revealing that CCDC32 inhibits assembly and requires membrane binding for complex formation.

The paper shows that the ATPase function of DPY-27 in C. elegans is essential for dosage compensation by enabling DNA binding, X-chromosome condensation, and H4K20me1 enrichment.

This study identifies novel separase regulators in C. elegans through an ENU mutagenesis screen, revealing mutations in pph-5 and hsp-90 that suppress a separase mutant phenotype.