Joel David Schwartz

This study links childhood exposure to different sources of PM2.5 pollution to changes in brain network connectivity across the U.S., with regional differences observed.

This study links outdoor air pollution exposure to increased perivascular space count and poorer cognition in preadolescents, suggesting brain clearance mechanisms may mediate pollution's neurotoxic effects.

This study conducts an epigenome-wide association study (EWAS) meta-analysis of wellbeing using DNA methylation data, finding no significant methylation sites but suggesting the need for larger studies.

The paper examines how ambient air pollution components and sources affect hippocampal structure and memory in pre-adolescents using data from the Adolescent Brain Cognitive Development Study.

The paper examines the relationship between outdoor air pollution and amygdala subregion volumes in early adolescents using ABCD Study data and finds associations between pollutants and specific subregion volume changes.

The paper examines how sleep duration and efficiency moderate the impact of prenatal and childhood air pollution on white matter integrity in adolescents.

This study examines how exposure to PM2.5 from biomass burning affects cortical microstructure development in adolescents using neuroimaging data.

The paper integrates DNA methylation with clinical risk factors to improve mortality prediction, identifying CpG biomarkers associated with all-cause, cardiovascular, and cancer mortality.

The paper develops a new statistical test, DACT, to detect mediation effects in genome-wide epigenetic studies and applies it to identify DNA methylation sites mediating smoking's effect on lung function.

The paper develops a DNA methylation-based blood test, DunedinPoAm, to quantify individual differences in the pace of biological aging and validates its association with aging-related outcomes across multiple studies.