Author

Juliette Bitard

Centre National de la Recherche ScientifiqueORCID 0000-0001-6626-3522Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

Jérôme E. RogeriD· 3 shared Muriel PerroniD· 3 shared Annaïg HamoniD· 2 shared Diana García-GarcíaiD· 2 shared Élodie-Kim Grellier· 2 shared Morgane LockeriD· 2 shared Albert Chesneau· 1 shared Christel MassoniD· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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3 shared papersNeuroscience

Jérôme E. RogerORCID

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3 papers

Recent·Most cited

NeurosciencebioRxivPreprintMay 16, 2022

Late-onset glaucoma in Yap conditional knockout mouse

Juliette Bitard, Élodie-Kim Grellier, Sophie Lourdel…·Muriel Perron

This study shows that Yap conditional knockout mice develop glaucoma features, including optic nerve degeneration and retinal ganglion cell death, highlighting YAP's role in preventing glaucoma.

DOI 10.1101/2022.05.16.492143

mouseexperimental assay

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NeuroscienceCell Death and DiseasePeer-reviewedAug 14, 2020

Yap haploinsufficiency leads to Müller cell dysfunction and late-onset cone dystrophy

Christel Masson, Diana García-García, Juliette Bitard…·Muriel Perron

This study investigates Yap haploinsufficiency in mice, revealing its role in Müller glia dysfunction and late-onset cone dystrophy in aged individuals.

18 citations

mousebulk RNA-seq

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Developmental BiologyCell ReportsPeer-reviewedMay 1, 2019

Linking YAP to Müller glia quiescence exit in the degenerative retina

Annaïg Hamon, Diana García-García, Divya Ail…·Muriel Perron

This study identifies YAP's role in Müller glia quiescence exit and regeneration by linking it to EGFR signaling and cell cycle re-entry in retinal injury.

104 citationstop 5%

mousezebrafish

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