NeurosciencebioRxivPreprint
A failure of β-amyloid physiological function due to genetic deletion of α7 nicotinic acetylcholine receptors induces an Alzheimer's disease-like pathology
Maria Rosaria Tropea, Domenica Donatella Li Puma, Marcello Melone…·Daniela Puzzo
This study shows that deleting α7 nicotinic acetylcholine receptors in mice leads to Alzheimer's-like pathology through increased amyloid-beta and tau abnormalities.
DOI 10.1101/2021.01.05.425382