Author

S. Tamir Rashid

Imperial College LondonORCID 0000-0001-6779-2506Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Adam Cryar· 1 shared Adriana OrdóñeziD· 1 shared Aleck W.E. Jones· 1 shared Bibek GooptuiD· 1 shared Charis‐Patricia Segeritz· 1 shared David A. LomasiD· 1 shared Graeme C. Smith· 1 shared Imran Haq· 1 shared

Activity by Year

2025-2025 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

Adam Cryar

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1 paper

Recent·Most cited

Cell BiologybioRxivPreprintSep 30, 2025

Both terminal misfolding and polymerisation contribute to disease-relevant phenotypic changes in cell models of alpha-1-antitrypsin deficiency-associated liver disease

Panayiota Stylianou, Kish R. Adoni, Mun Peak Nyon…·Bibek Gooptu

This study shows that both terminal misfolding and polymerisation of Z 1-antitrypsin contribute to liver disease phenotypes, with terminal misfolding driving pro-fibrotic responses and metabolic changes.

DOI 10.1101/2025.09.30.679226

humanproteomics

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