Natalie L. Truby

This study shows that altering TRIM28 function in the mouse prefrontal cortex leads to transposable element derepression and social behavior deficits, which can be reversed by interferon cytokines.

This study finds that transposable elements are downregulated in the brains of individuals with Major Depressive Disorder, particularly in the orbitofrontal cortex, and this effect varies by age and sex.

This study identifies ZFP189's role in regulating transposable elements in medium spiny neurons, showing how TE activation or repression in specific neuronal subtypes influences cocaine-related behaviors and gene expression.

The paper investigates whether elevated H1x protein levels in the ventral hippocampus cause stress-related behavioral adaptations, finding no significant behavioral effects from H1x overexpression.

This study shows that a synthetic zinc finger transcription factor modulates social behaviors by regulating transposable elements and immune response in the prefrontal cortex of mice.