Rachael L. Neve

This study shows that altering TRIM28 function in the mouse prefrontal cortex leads to transposable element derepression and social behavior deficits, which can be reversed by interferon cytokines.

This study shows that E2F3a overexpression in D1-MSNs increases cocaine reward in both male and female mice, while knockdown has opposite effects, with no impact in D2-MSNs.

The paper investigates whether elevated H1x protein levels in the ventral hippocampus cause stress-related behavioral adaptations, finding no significant behavioral effects from H1x overexpression.

This study shows that a synthetic zinc finger transcription factor modulates social behaviors by regulating transposable elements and immune response in the prefrontal cortex of mice.

This study identifies RXR as a key regulator of drug reward sensitivity in the nucleus accumbens through transcriptional control of neuronal excitability in mice.

The paper develops viral tools for conditional and transneuronal gene expression in zebrafish to study neuronal circuits.

This paper identifies a novel vCA1[->]pPVN circuit that modulates both learned and innate fear by regulating neuronal activity in the PAG.

This study identifies a PACAPergic microcircuit in the amygdala that modulates contextual fear in a sex-dependent manner, with distinct effects on fear acquisition, generalization, and extinction.

The paper identifies Zfp189 as a key driver of a transcriptional network promoting stress resilience in prefrontal cortex neurons through CREB regulation.

This study identifies distinct sub-systems within the dorsal raphe serotonin system with different connectivity, physiological responses, and behavioral functions.