Author

Frank Bridoux

Centre National de la Recherche ScientifiqueORCID 0000-0002-2005-8424Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:protein structure· 1

Frequent Co-Authors

Arnaud JaccardiD· 2 shared Christophe SiraciD· 2 shared Gemma Martinez-RivasiD· 2 shared Sébastien BenderiD· 2 shared Alan C. StuckeyiD· 1 shared Alessio LampisiD· 1 shared Alexa Rinsant· 1 shared Alexander CarpinteiroiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersPathology

Arnaud JaccardORCID

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2 papers

Recent·Most cited

Preclinical characterization of AT-03, a novel Serum Amyloid P fusion protein that demonstrates pan-amyloid binding and removal

Christophe Sirac, Gilles Roussine Codo, Arnaud Jaccard, Sébastien Bender, Gemma Martinez-Rivas, Frank Bridoux, Marie Clavel, Camille Villesuzanne, Oussama Taoui, Claire Carrion, Gregory Bell, Spencer D. Guthrie, Suganya Selvarajah, Andrew Vick, Stephen J. Kennel, , Jonathan S. Wall

This paper characterizes AT-03, a novel fusion protein that binds and removes various amyloid types, showing therapeutic efficacy in a mouse model.

bioRxiv · Dec 25, 2025 · PreprintPathology

A mouse model of cardiac AL amyloidosis unveils mechanisms of tissue accumulation and toxicity of amyloid fibrils

Gemma Martinez-Rivas, Maria Victoria Ayala, Sébastien Bender, Gilles Roussine Codo, Karolina Weronika Świderska, Alessio Lampis, Laura Pedroza, Melisa Merdanovic, Pierre Sicard, Émilie Pinault, Laurence Richard, Francesca Lavatelli, Sofia Giorgetti, Diana Canetti, Alexa Rinsant, , Christophe Sirac

This paper develops a mouse model of cardiac AL amyloidosis that reproduces human disease features, including amyloid deposits and cardiac dysfunction, and identifies a role for partial LC degradation in fibril formation.

Nature Communications · Mar 27, 2025Peer-reviewedPathology