Simon J. Foster

This paper investigates the morphological transformation of Helicobacter pylori from helical to coccoid form using microscopy techniques, revealing it occurs without a U-shaped intermediate and involves peptidoglycan thinning and membrane bulging.

The paper shows that ClpX chaperone and a hypermorphic FtsA variant compensate for each other in Staphylococcus aureus cell division by modulating FtsA interactions and septum synthesis.