Camilla Henriksen

This study identifies a temperature-sensitive regulatory mechanism in Staphylococcus aureus involving an RNA and ClpXP protease that controls cell division hydrolases.

This study identifies Spx as a stress regulator that enhances β-lactam resistance and reduces susceptibility to last-line antibiotics in MRSA through a redox-independent mechanism.

The paper shows that ClpX chaperone and a hypermorphic FtsA variant compensate for each other in Staphylococcus aureus cell division by modulating FtsA interactions and septum synthesis.