Ingrid Brust‐Mascher

This study shows that neonatal EPEC infection disrupts the microbiota-gut-brain axis via NOD1 signaling, leading to long-term neuroimmune and behavioral deficits, and identifies probiotic muropeptides as potential therapeutics.

This study shows that the sympathetic nervous system enhances immune responses to enteric bacterial infections by modulating IFN{gamma} production in CD4+ T-cells via alpha-adrenergic receptors.

This study identifies a neuroimmune circuit activated by afferent vagus nerve stimulation that reduces acute lung inflammation through epinephrine and β2-adrenergic receptor signaling.

This study shows that blocking Substance P receptor signaling reduces inflammation and prevents maladaptive responses during enteric bacterial infection without affecting host protection.