Author

Shikha Kataria

Wellcome Sanger InstituteORCID 0000-0001-7889-3070Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:scRNA-seq· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1 Research Focus:variant effect prediction· 1

Frequent Co-Authors

Aleksander M. Gontarczyk· 2 shared Andrew Roger BassettiD· 2 shared Adib Miraki FeriziD· 1 shared Andrew J. WatersiD· 1 shared Caia S. DominicusiD· 1 shared Claudia FengiD· 1 shared D. Adams· 1 shared Daniel P. GittermaniD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersGenomics

Aleksander M. Gontarczyk

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2 papers

Recent·Most cited

GenomicsbioRxivPreprintOct 14, 2026

Transcriptomic and phenotypic characterization of Alzheimer's disease risk genes in neurons and astrocytes highlights shared and cell type specific cellular mechanisms

Caia S. Dominicus, Diego Peretti, Geraldine Rodschinka…·Andrew Roger Bassett

The paper uses single-cell transcriptomics and phenotypic analysis of gene knockouts in hiPSC-derived neurons and astrocytes to identify cell-type specific and shared mechanisms of Alzheimer's disease risk genes.

DOI 10.64898/2026.10.13.758778

humanscRNA-seq

𝕏 PostBluesky

GenomicsbioRxivPreprintJul 4, 2026

Saturation-seq integrates single-cell saturation genome editing and RNA-seq to quantify NFE2L2 (NRF2) variant effects

Magdalena E Strauß, Andrew J. Waters, Holly Roberston…·D. Adams

Saturation-seq combines genome editing and single-cell RNA-seq to quantify variant effects on NFE2L2, accurately distinguishing pathogenic from benign variants.

DOI 10.64898/2026.06.30.735631

humanscRNA-seq

𝕏 PostBluesky