Author

Paul Davies

University of DundeeORCID 0000-0001-8022-2719Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Paul Davies3 papers · Animal and Plant Health Agency

Biology Disciplines

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Developmental Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Jacqueline Kim DaleiD· 2 shared Philip J. MurrayiD· 2 shared Adam Hetherington· 1 shared David M. A. MartiniD· 1 shared Francesca A. CarrieriiD· 1 shared Hedda A. MeijeriD· 1 shared Izzah N. Hussein· 1 shared Jess M. Rae· 1 shared

Activity by Year

2018-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersDevelopmental Biology

Jacqueline Kim DaleORCID

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2 papers

Recent·Most cited

Developmental BiologyGenes & DevelopmentPeer-reviewedSep 1, 2025

NOTCH1 S2513 is critical for the regulation of NICD levels impacting the segmentation clock in hiPSC-derived PSM cells and somitoids

Hedda A. Meijer, Adam Hetherington, Sara Johnson…·Jacqueline Kim Dale

This study shows that a NOTCH1 mutation affects NICD stability and disrupts segmentation clock function in hiPSC-derived PSM cells and somitoids.

6 citations

humanbulk RNA-seq

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Developmental BiologybioRxivPreprintJan 9, 2018

CDK1 and CDK2 regulate phosphorylation-dependent NICD1 turnover and the periodicity of the segmentation clock

Francesca A. Carrieri, Philip J. Murray, Paul Davies, Jacqueline Kim Dale

This study identifies CDK1 and CDK2 as regulators of NICD1 phosphorylation and turnover, showing their role in controlling segmentation clock periodicity.

2 citations

mouseproteomics

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