Jie‐Min Jia

This study shows that neurovascular coupling in the basolateral amygdala bidirectionally modulates stress-induced negative emotions through manipulations in freely moving mice.

This study shows that myeloid cell-driven transdifferentiation of endothelial cells into pericytes helps restore BBB function and brain repair after stroke.

The paper develops a method to analyze high-resolution vasomotion in mouse cerebrovasculature, revealing new physiological features and changes in vascular networks after stroke.

This study identifies Ca2+ oscillations in vascular smooth muscle cells as critical for myogenic vasomotion and shows that restoring mitochondrial-ER contacts can improve reperfusion after ischemic stroke.

The paper identifies Netrin4 as a factor essential for adult sympathetic neuron survival, showing it promotes protein synthesis and prevents necroptosis in specific neurons.

This study shows that ErbB inhibition in oligodendrocytes disrupts myelination and aerobic glycolysis, impairing working memory in mice.

This study shows that blood vessel regression in the brain reduces neuronal activity by impairing mitochondrial metabolism and glutamate production.