Author

Elisa Gorostieta-Salas

Salk Institute for Biological StudiesORCID 0000-0001-8485-705XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Bioengineering· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Assay:metabolomics· 1

Frequent Co-Authors

Fred H. GageiD· 2 shared Adam C. FarsheediD· 1 shared Angelo D' Alessandro· 1 shared Ayk Petrosyan· 1 shared Cat Siler· 1 shared Colin C. AndersoniD· 1 shared Crystal LuiD· 1 shared Douglas Galasko· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersBioengineering

Fred H. GageORCID

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2 papers

Recent·Most cited

BioengineeringbioRxivPreprintOct 28, 2026

Growing Macroscopic Functional Human Tissues on 3D Printed Peptide Hydrogels

Adam C. Farsheed, Jonathan T. Makhoul, Justin Liu…·Fred H. Gage

This study demonstrates growing macroscopic functional human tissues on 3D-printed peptide hydrogels, enabling centimeter-scale neural organoids and directional axonal communication.

DOI 10.64898/2026.10.26.761928

humanbulk RNA-seq

𝕏 PostBluesky

NeurosciencebioRxivPreprintOct 1, 2026

Nuclear acetyl-CoA accumulation drives reversible RBP hyperacetylation and transcriptome-wide splicing shifts in age-equivalent Alzheimer's disease neurons

Larissa Traxler, Oliver Borgogno, Kylie Champion…·Jérôme Mertens

This study shows that nuclear acetyl-CoA accumulation in Alzheimer's disease neurons causes RBP hyperacetylation and splicing shifts, which can be reversed by inhibiting ACLY.

DOI 10.64898/2026.09.26.754222

humanscRNA-seq

𝕏 PostBluesky