Author

Cristina de Guzman Strong

Henry Ford HospitalORCID 0000-0001-8540-9611Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Bioinformatics· 1 Developmental Biology· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Organism:human· 3 Approach:experimental assay· 2 Organism:mouse· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Assay:DNA methylation· 1

Frequent Co-Authors

Alina D. SchmidtiD· 2 shared Mary Elizabeth MathyeriD· 2 shared A. Quiggle· 1 shared Albert M. LeviniD· 1 shared Ali J. MarianiD· 1 shared Antonietta FrancoiD· 1 shared Avner ShemeriD· 1 shared Daniel P. KellyiD· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersDevelopmental Biology

Alina D. SchmidtORCID

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3 papers

Recent·Most cited

BioinformaticsbioRxivPreprintSep 7, 2026

Keloid transcriptomics reveal heterogeneity in fibroblast subtype enrichment, gene expression, and immune cell responses

Jonathan J. Panzer, Mingming Pan, Mrudula Nair…·Lamont R. Jones

The paper identifies cell type-specific gene expression and immune cell associations in keloid tissue using bulk RNA-Seq and single sample gene set enrichment analysis.

DOI 10.64898/2026.09.02.747090

humanbulk RNA-seq

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Developmental BiologyeLifePeer-reviewedSep 27, 2023

A human mitofusin 2 mutation causes mitophagic cardiomyopathy

Antonietta Franco, Jiajia Li, Daniel P. Kelly…·Gerald W. Dorn

The paper identifies a mitofusin 2 mutation (R400Q) that causes mitophagic cardiomyopathy by impairing mitochondrial fusion and Parkin recruitment, distinct from its role in CMT2A.

31 citations

humanmouse

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GeneticsbioRxivPreprintNov 5, 2019

An enhancer:involucrin regulatory module impacts human skin barrier adaptation out-of-Africa and modifies atopic dermatitis risk

Mary Elizabeth Mathyer, Erin A. Brettmann, Alina D. Schmidt…·Cristina de Guzman Strong

The paper identifies an enhancer-IVL regulatory module that evolved in humans and is associated with skin barrier adaptation and atopic dermatitis risk.

1 citation

humanmouse

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