Author

Ariel E. Timkovich

University of Colorado AnschutzORCID 0000-0001-8652-5016Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:human· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:gene regulatory network inference· 1 Assay:protein sequence· 1

Frequent Co-Authors

Angela L. RachubinskiiD· 2 shared Joaquin Maximiliano EspinosaiD· 2 shared Kelly D. SullivaniD· 2 shared Kyndal A. Schade· 2 shared Lauren N. DunniD· 2 shared Matthew D. GalbraithiD· 2 shared Brian F. NiemeyeriD· 1 shared Brian Niemeyer· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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2 shared papersGenomics

Angela L. RachubinskiORCID

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2 papers

Recent·Most cited

A Trisomy 21 Model Atlas reveals conserved dosage effects and context-specific transcriptional responses across mouse and human models of Down syndrome

Karen Rossmassler, Ariel E. Timkovich, Brian Niemeyer, Kyndal A. Schade, Lauren N. Dunn, Macallister C. Harris, R Adam Harris, Srija Chillamcherla, Angela L. Rachubinski, Joaquin Maximiliano Espinosa, Kelly D. Sullivan, Matthew D. Galbraith

The paper presents a Trisomy 21 Model Atlas comparing mouse and human models to show conserved dosage effects and context-specific transcriptional responses across tissues and developmental stages.

bioRxiv · Oct 14, 2026 · PreprintGenomics

Altered Hepatic Metabolism in Down Syndrome

Lauren N. Dunn, Brian F. Niemeyer, Neetha Paul Eduthan, Kyndal A. Schade, Katherine A. Waugh, Chrisstopher Brown, Angela L. Rachubinski, Ariel E. Timkovich, David J. Orlicky, Matthew D. Galbraith, Joaquin Maximiliano Espinosa, Kelly D. Sullivan

The paper reports altered hepatic metabolism in Down syndrome using multi-omic analysis of plasma and mouse models, showing increased bile acids and liver dysfunction that can be modulated by diet.

Cell Reports · Jan 1, 2026Peer-reviewedGenetics