Lipid Activation of a Thalamic GPCR Extends Working Memory Time-scales
James Newton Brandt, Alessandra Bonito-Oliva, Navid Paknejad…·Priyamvada Rajasethupathy
This study identifies a lipid signaling mechanism involving AEA-Gpr12 in thalamus that extends working memory duration by suppressing cAMP and supporting sustained neural activity in PFC.