Author

Fabrizio Pontarelli

Sanofi (France)ORCID 0000-0001-8959-9918Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Organism:human· 1 Organism:mouse· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Dimitry OfengeimiD· 2 shared Joseph Gans· 2 shared Matija ZeliciD· 2 shared Sean K. RyaniD· 2 shared Timothy R. HammondiD· 2 shared Anna S. Blazier· 1 shared Bailin ZhangiD· 1 shared Bailin ZhangiD· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersNeuroscience

Dimitry OfengeimORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintApr 12, 2024

Glial state changes and neuroinflammatory RIPK1 signaling are a key feature of ALS pathogenesis

Matija Zelic, Anna S. Blazier, Fabrizio Pontarelli…·Timothy R. Hammond

This study identifies glial state changes and RIPK1 signaling as key features of ALS pathogenesis, showing that blocking RIPK1 delays disease onset and modulates glial responses.

2 citations

mousescRNA-seq

𝕏 PostBluesky

NeurosciencebioRxivPreprintNov 2, 2021

Microglia ferroptosis is prevalent in neurodegenerative disease and regulated by SEC24B

Sean K. Ryan, Matija Zelic, Yingnan Han…·Timothy R. Hammond

The paper shows microglia are susceptible to ferroptosis in neurodegenerative diseases and identifies SEC24B and small molecules that regulate this process.

4 citations

humanbulk RNA-seq

𝕏 PostBluesky