Glutamate signaling and neuroligin/neurexin adhesion play opposing roles that are mediated by major histocompatibility complex I molecules in cortical synapse formation
Gabrielle L. Sell, Stephanie L. Barrow, A. Kimberley McAllister
Author
University of California, DavisORCID 0000-0001-9177-9889Unclaimed ORCID profile · follow to get notified of new preprints
7 papers
Recent·Most cited
Gabrielle L. Sell, Stephanie L. Barrow, A. Kimberley McAllister
Shekib A. Jami, Scott Cameron, Jonathan M. Wong…·John A. Gray
The paper investigates the relationship between NMDA receptor hypofunction and GABAergic synapse loss in SRKO mice, finding reduced inhibitory synapses and increased excitation-inhibition imbalance.
Nicholas F. Page, Michael J. Gandal, Myka L. Estes…·Daniel H. Geschwind
This study identifies transcriptomic changes in non-human primate brains exposed to maternal immune activation, revealing alterations in retrotransposition, synaptic connectivity, and myelination.
Roza Vlasova, Ana‐Maria Iosif, Amy M. Ryan…·Melissa D. Bauman
This study examines the effects of maternal immune activation on neurobehavioral development in rhesus monkey offspring, finding reduced gray matter volume in specific brain regions.
César P. Canales, Myka L. Estes, Karol Cichewicz…·Alex S. Nord
This study identifies transcriptomic and neuroanatomical changes in mouse embryos exposed to maternal immune activation, revealing altered neurodevelopmental timing and cortical development.
Myka L. Estes, Kathleen Farrelly, Scott Cameron…·A. Kimberley McAllister
This study identifies variability factors in maternal immune activation models and shows baseline immune responsiveness predicts offspring outcomes.
Myka L. Estes, Bradford M. Elmer, Cameron Carter, A. Kimberley McAllister
This study shows maternal immune activation causes age-specific changes in cytokine receptor expression in offspring's frontal cortex, with potential implications for neuropsychiatric disorders.